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Thrombolysis

THEIA Trial: IV Alteplase vs Oral Aspirin for Acute Non-Arteritic CRAO within 4.5 Hours

In adults with acute non-arteritic central retinal artery occlusion (Snellen <20/400) within 4.5h of symptom onset, does IV alteplase 0.9 mg/kg improve visual acuity at 1 month compared with 300 mg oral aspirin? First phase 3 RCT of IV thrombolysis for CRAO.

Préterre et al. (Lancet Neurology 2025) · doi:10.1016/S1474-4422(25)00308-4 · 70 patients

Population

Included

  • Adults aged 18 years or older
  • Sudden severe persistent monocular vision loss (Snellen <20/400; >1.3 LogMAR)
  • Suspected non-arteritic acute CRAO confirmed by ophthalmologist (fundoscopy or non-mydriatic retinophotography showing diffuse retinal pallor, macular cherry red spot, vessel attenuation, or visible emboli)
  • Relative afferent pupillary defect on the affected side
  • Treatment startable within 4.5 hours of symptom onset
  • No clinical or laboratory evidence of giant cell arteritis
  • No clinical or radiological evidence of stroke within the prior 3 months (except asymptomatic punctate or small DWI lesions)
  • Baseline CT or MRI brain imaging available

Excluded

  • Minor visual acuity deficits or rapid pre-treatment improvement
  • Unknown or uncertain symptom onset time
  • Isolated branch retinal artery occlusion without clinically significant vision loss
  • CRAO with foveal sparing due to cilioretinal artery (no foveal ischaemia)
  • Current use of anticoagulant medication
  • Standard contraindications to IV alteplase (active bleeding, recent major surgery, BP >185/110)

Primary Outcome

Visual acuity improvement ≥0.3 LogMAR (~3 Snellen lines / 15 ETDRS letters) at 1 month

Alteplase Group (19 of 29)
66 / 100
Aspirin Group (13 of 27)
48 / 100

Visual acuity ≥0.3 LogMAR gain at 1 month

Risk ratio aOR 1.1095% CI 0.07–18.39p = 0.95

Unadjusted risk difference +17.4 pp (95% CI -11.8 to +46.5). Trial was powered for a 30 pp difference; observed aspirin response of 48% (vs assumed 10%) made the trial underpowered for the smaller real difference. p=0.95 reflects no statistical signal — not evidence of no effect.

Study Arms

Agent
Alteplase
Dose
0.9 mg/kg of bodyweight, maximum 90 mg
Route
Intravenous
Frequency
Single dose within 4.5 h of symptom onset
Duration
10% of the dose as a bolus, remainder infused over 1 h
Co-interventions
Double-dummy: also received one oral dose of placebo. Standard stroke-unit care and secondary prevention per European Stroke Organisation guidelines. Anticoagulants, additional thrombolytic agents, and antiplatelet drugs were not permitted during the first 24 h.

Patient-blinded and assessor-blinded double-dummy design.

Safety

Symptomatic intracranial haemorrhage (ECASS II)

Alteplase Group (19 of 29)

0%

Aspirin Group (13 of 27)

0%

Zero symptomatic ICH in either arm. One asymptomatic intracranial haemorrhage (15 mm right parietal haematoma) detected on day 1 CT in the alteplase group. Not present on initial MRI, no neurological consequences. No symptomatic haemorrhages or major bleeding related to study treatment in either arm. This is much lower than the typical ~6% sICH rate with IV alteplase in acute ischaemic stroke, likely reflecting the absence of established ischaemic brain lesion in CRAO.

Major extracranial bleeding related to treatment

Alteplase Group (19 of 29)

0%

Aspirin Group (13 of 27)

0%

Zero major extracranial bleeds related to study treatment in either arm. One after-acute-phase gastrointestinal bleed (colon polyps, requiring transfusion) in the aspirin group attributed to secondary-prevention antithrombotic therapy, not the acute study drug.

Serious adverse events after acute phase (%)

Alteplase Group (19 of 29)

14%

Aspirin Group (13 of 27)

17%

Alteplase 5/35 (14%) vs aspirin 6/35 (17%). Most were extra-ocular ischaemic events (carotid endarterectomy in 6, pyelonephritis in 5, melena on colon polyps, and one manic episode in a known bipolar patient). None related to acute study treatment.

Trial Design

Type

  • Phase 3, multicentre randomised controlled trial
  • Double-dummy, patient-blinded and assessor-blinded
  • 1:1 allocation (alteplase + oral placebo vs aspirin + IV saline placebo)
  • Conducted across 16 French stroke units
  • Binary-superiority primary; underpowered per investigators

Timeline

Enrolled 8 June 2018 – 2 October 2023; published Lancet Neurology November 2025.

N

70

Enrollment

70 patients (35 alteplase / 35 aspirin) at 16 French stroke units. Enrolled June 2018 to October 2023. Phase 3 double-dummy patient- and assessor-blinded RCT. Primary endpoint: visual acuity ≥0.3 LogMAR gain at 1 month. Underpowered per investigators. Published Lancet Neurology November 2025.

ClinicalTrials.gov

NCT03197194

Bedside Pearl

THEIA (Lancet Neurology 2025): first phase 3 RCT of IV alteplase 0.9 mg/kg vs aspirin 300 mg within 4.5h of non-arteritic CRAO. Visual acuity improvement at 1 month 66% vs 48% (adjusted OR 1.10, p=0.95); directionally favouring alteplase but not significant; trial underpowered at N=70. Zero symptomatic ICH in either arm. Result does not prove alteplase ineffective; it underscores the need for the planned IPD meta-analysis (THEIA + TenCRAOS + REVISION). Pre-THEIA guidance (Mac Grory AHA/NANOS 2021): IV alteplase "may be considered" for disabling CRAO meeting criteria. THEIA does not change that.

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