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ANNEXA-I Trial: Andexanet Alfa for Factor Xa Inhibitor-Associated Acute Intracerebral Hemorrhage

In adults with acute intracerebral hemorrhage on apixaban, rivaroxaban, or edoxaban within 15 hours of last dose (hematoma 0.5–60 mL, NIHSS ≤35, GCS ≥7), does andexanet alfa improve a composite hemostatic-efficacy surrogate at 12 hours compared with usual care (85.5% of which was 4F-PCC)? First randomized trial of an FXa-inhibitor antidote specifically in acute ICH.

Connolly SJ et al. (NEJM 2024;390(19):1745-1755) · doi:10.1056/NEJMoa2313040 · 530 patients

Population

Included

  • Adults with acute intracerebral hemorrhage confirmed on CT or MRI within 2 hours before randomization
  • Hematoma volume 0.5 to 60 mL on baseline imaging
  • Most recent FXa inhibitor (apixaban, rivaroxaban, or edoxaban) dose within 15 hours before randomization
  • NIHSS at most 35
  • GCS at least 7 at time of consent
  • Time from symptom onset to baseline scan within 6 hours (per amendment; originally 12 hours)

Excluded

  • Hematoma volume greater than 60 mL
  • Planned neurosurgical intervention within 12 hours of enrollment
  • Thrombotic event within 2 weeks before randomization
  • Prior andexanet exposure
  • Subdural or subarachnoid hemorrhage as primary bleeding site (after protocol amendment)
  • GCS below 7 or NIHSS above 35

Primary Outcome

Composite hemostatic efficacy at 12 h: hematoma volume change ≤35%, NIHSS rise <7, no rescue therapy 3–12 h

Andexanet AlfaBetter outcome
67 / 100
Usual Care (85.5% received 4F-PCC)
53 / 100

Hemostatic efficacy at 12 h

Risk ratio +13.4 pp95% CI 4.6–22.2p = 0.003
NNT8for the composite hemostatic surrogate at 12 h (NOT functional independence or survival)

Composite was driven by hematoma-volume control; NIHSS-change and rescue-therapy components did not differ. Trade-off: ischemic stroke 6.5% vs 1.5% (+5.0 pp, NNH 20); any thrombotic event 10.3% vs 5.6% (P=0.048, NNH 22). No functional benefit (exploratory mRS 0–3 at 30 days: 28.0% vs 31.0%) and no mortality benefit (27.8% vs 25.5%, P=0.51). DSMB-halted early at pre-specified interim — effect size may be overestimated.

Study Arms

Agent
Andexanet alfa, a modified recombinant inactive form of human factor Xa that binds and sequesters factor Xa inhibitor molecules, rapidly reducing anti-factor Xa activity and restoring thrombin generation
Dose
A high-dose bolus or a low-dose bolus followed by a continuous infusion. The use of a high-dose or low-dose bolus was in accordance with the label approved by the Food and Drug Administration and was based on the type, the amount, and the timing of the most recent dose of the factor Xa inhibitor that was received. The andexanet formulation included 500 mg of mannitol per vial.
Route
IV
Frequency
Bolus over the course of 15 to 30 minutes followed by a continuous infusion over the course of 2 hours
Duration
Approximately 2 hours (bolus plus continuous infusion)
Co-interventions
Usual supportive care for acute intracerebral hemorrhage. Prothrombin complex concentrate was not given to the andexanet group as part of the assigned treatment

Dose/administration verbatim from Connolly NEJM 2024 p.1747 (Randomization and Treatments). 78.1% of andexanet patients received the low-dose regimen and 20.1% received high-dose. Anti-factor Xa activity fell by a median of 94.5% at the 1-to-2-hour nadir. Open-label assignment with blinded outcome adjudication.

Safety

30-day mortality

Andexanet Alfa

27.8%

Usual Care (85.5% received 4F-PCC)

25.5%

30-day mortality 73 of 263 (27.8%) andexanet vs 68 of 267 (25.5%) usual care. Adjusted difference 2.5 percentage points (95% CI -5.0 to 10.0), P=0.51. No mortality benefit; trial not powered to detect a mortality difference. Source: Connolly SJ et al., NEJM 2024, Table 3 p.1753.

Ischemic stroke within 30 days

Andexanet Alfa

6.5%

Usual Care (85.5% received 4F-PCC)

1.5%

Ischemic stroke within 30 days: 17 of 263 (6.5%) andexanet vs 4 of 267 (1.5%) usual care. Increase 5.0 percentage points (95% CI 1.5-8.8). NNH approximately 20 for ischemic stroke. Source: Connolly SJ et al., NEJM 2024, Table 3 p.1753. Note: hemorrhagicStroke field repurposed here to surface the dominant thrombotic-event component; this is an ISCHEMIC stroke signal in ANNEXA-I.

Any thrombotic event within 30 days

Andexanet Alfa

10.3%

Usual Care (85.5% received 4F-PCC)

5.6%

Any thrombotic event within 30 days: 27 of 263 (10.3%) andexanet vs 15 of 267 (5.6%) usual care. Adjusted increase 4.6 percentage points (95% CI 0.1-9.2), P=0.048. Assessed in the extended population (530 patients including those enrolled after database lock for the interim analysis). Source: Connolly SJ et al., NEJM 2024, Table 3 p.1753.

Trial Design

Type

  • Multicenter multinational open-label RCT with blinded outcome adjudication
  • 1:1 randomization stratified by intention to use PCC and (after amendment) time-to-baseline-scan
  • Pre-specified interim analysis at 450 patients with significance threshold P<0.031
  • DSMB-halted at interim for hemostatic efficacy (May 2023, 452 patients analyzed)
  • Hierarchical testing to preserve family-wise alpha at 5%

Timeline

Enrolled June 2019 to May 2023; stopped early for efficacy at pre-specified interim analysis

N

530

Enrollment

530 patients in the extended safety population (263 andexanet / 267 usual care). Primary efficacy at pre-specified interim: 452 patients (224 andexanet / 228 usual care). 131 sites in 23 countries. Enrolled June 2019 to May 2023. Multicenter multinational open-label RCT with blinded outcome adjudication. Originally planned for 900 patients; DSMB halted at interim (May 2023) after hemostatic-efficacy threshold P<0.031 crossed. Published NEJM 2024.

ClinicalTrials.gov

NCT03661528

Bedside Pearl

For acute intracerebral hemorrhage on an FXa inhibitor (apixaban, rivaroxaban, edoxaban) within 15 hours of last dose, andexanet improved a composite hemostatic surrogate (hematoma expansion, NIHSS, rescue therapy) at 12 h from 53.1% to 67.0% (NNT 8). Anti-FXa activity dropped 94.5% vs 26.9%. The cost: ischemic stroke 6.5% vs 1.5% (NNH 20) and any thrombotic event 10.3% vs 5.6% (NNH 22). No functional benefit or mortality benefit at 30 days. Quote both NNT and NNH when consenting; if patient has high cardioembolic baseline (atrial fibrillation, recent thrombotic event) the trade-off shifts. 4F-PCC remains the comparator that 85.5% of usual-care patients actually received.

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